1、

In addition , apoptosis did occur in AEC isolated from TNF KO mice following LPS stimulation.

在TNF基因剔除(knockout,KO)小鼠,LPS仍 可明显诱导AEC凋亡.

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2、

ACP, GOT, GPT, CHE, CK, LPS and LDH activities had little correlation with fiber digestibility and Met, Cys digestibility ( P & gt; 0.05 ).

酸性磷酸酶(ACP) 、 谷草转氨酶(GOT) 、 谷丙转氨酶(GPT) 、 胆碱酯酶(CHE) 、 肌酸激酶(CK) 、 脂肪激酶(LPS)和乳酸脱氢酶(LDH)的活性与营养物的消化率呈弱相关 ( P & gt; 0.05 ).

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3、

CONCLUSION: LPS induces liver IL-6 expression , which might activate BEC proliferation through STAT 3 pathway.

结论: LPS刺激导致肝组织IL-6分泌增加, 后者可能通过STAT3介导BEC增殖.

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4、

Conclusion LPS could involve bone tissue in inflammation and inhibit the differentiation of osteoblast.

结论LPS刺激骨组织发生炎症反应、抑制成骨细胞分化.

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Conclusion: The role of LPS in caries-induced reparative dentin might be a significant determinant.

结论: 提示适度的LPS在牙髓对龋病的防御反应过程中起着一定调节作用.

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6、

Conclusion LPS can stimulate expression of ESM 1 in endothelial cells.

结论 LPS对ECV304中ESM1的表达有促进作用.

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7、

There were 76 ODPs and 285 LPs covering the HKSAR.

年底时,全港共有76份发展大纲图和285份发展蓝图.

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8、

Conclusion LPS can inhibit the apoptosis of PMNs.

结论LPS对PMNs的凋亡有抑制作用.

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9、

PurposeTo study extraction, purification and physicochemical analysis of Laminaria polysaccharide sulfate ( LPS ).

目的研究海带硫酸多糖 ( LPS ) 的提取制备及其组分的纯化和理化性质.

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10、

In this reaction, PS was decomposed into polysilane of low molecu-lar weight ( LPS ) containing Si-C skeleton and Si-H bonds.

在这一反应中, PS裂解为含Si—H键的低分子聚硅烷 ( LPS ).

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11、

The ultrastructure of Clara cells damaged significantly 24 h post LPS injection.

LPS致伤后24h,Clara细胞的超微结构明显受损.

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12、

In group NL, the serum ACE increased at 12 h ahd 24 h ( P < 0.05 ) after LPS injection.

NL组于12、24hACE明显高于对照组 ( P<0.05 ).

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13、

ACP, GOT, GPT, CHE, CK, LPS and LDH activities had little correlation with fiber digestibility and Met, Cys digestibility ( P & gt; 0.05 ).

酸性磷酸酶(ACP) 、 谷草转氨酶(GOT) 、 谷丙转氨酶(GPT) 、 胆碱酯酶(CHE) 、 肌酸激酶(CK) 、 脂肪激酶(LPS)和乳酸脱氢酶(LDH)的活性与营养物的消化率呈弱相关 ( P & gt; 0.05 ).

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14、

Conclusion: Haizhu Yigan capsule has remarkable protective effect against the immunological liver injury induced by BGG in addition with LPS in the immunological hepatitis model.

结论:海珠益肝胶囊对BCG加LPS诱导小鼠产生免疫性肝炎的模型免疫性肝损伤具有显著的保护作用。

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The avidity of serum IgG against Pg LPS was measured by ELISA with thiocyanate elution before and after periodontal treatment.

治疗前后,实验检测所有受试者血清抗Pg抗体亲和力水平。亲和力通过二乙醇胺分离ELISA测定。

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16、

Studies on ELISA for Detection of Bovine Brucellosis with Heating-phenol Extracted LPS Antigen

牛布氏杆菌病热酚法脂多糖抗原酶联免疫吸附试验(ELISA)的研究

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17、

In group NL, the serum ACE increased at 12 h ahd 24 h ( P < 0.05 ) after LPS injection.

NL组于12、24hACE明显高于对照组 ( P<0.05 ).

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18、

Conclusion IL-12, IL-2 and LPS may promote lck gene express in renal tubular epithelial cells and only IL-12 can induce p38 phosphorylation through lck uniquely, and they can play biologic role in renal tubular epithelial cells via lck/ p38 signaling pathways.

结论IL-12、IL-2、LPS促进肾小管上皮细胞中lck基因表达,并只有IL-12唯一通过lck诱导p38磷酸化,它们皆可通过lck/p38信号传递途径参与对肾小管上皮细胞发挥生物学作用。

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Study of Soft Tissue Infectious in Skin and Dynamic Monitoring of LPS Level

皮肤软组织感染病人与血浆脂多糖

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Study of Soft Tissue Infectious in Skin and Dynamic Monitoring of LPS Level

皮肤软组织感染病人与血浆脂多糖

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